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GeneQ18038193· pop 6· linked from 4 articles

Also known as OPT, opticin

Opticin is a protein that in humans is encoded by the OPTC gene.

Gene data

OPTC
Name
opticin
Type
protein-coding
Position
203,494,118–203,509,238 (+)
Aliases
OPT
RefSeq RNA
NM_014359.4
RefSeq protein
NP_055174.1

Opticin belongs to class III of the small leucine-rich repeat protein (SLRP) family. Members of this family are typically associated with the extracellular matrix. Opticin is present in significant quantities in the vitreous of the eye and also localizes to the cornea, iris, ciliary body, optic nerve, choroid, retina, and fetal liver. Opticin may noncovalently bind collagen fibrils and regulate fibril morphology, spacing, and organization. The opticin gene is mapped to a region of chromosome 1 that is associated with the inherited eye diseases age-related macular degeneration (AMD) and posterior column ataxia with retinosa pigmentosa (AXPC1). [provided by RefSeq, Jul 2008].

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Gene · Ensembl

opticin

Symbol
OPTC
Biotype
Protein coding
Organism
Homo sapiens
Location
1:203,494,118-203,509,238
Strand
Forward (+)
Assembly
GRCh38
View on Ensembl →

via Ensembl · EMBL-EBI

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Encyclopedic overview

2 sections
Contents
  • References
  • Further reading

Opticin is a protein that in humans is encoded by the OPTC gene.

Opticin belongs to class III of the small leucine-rich repeat protein (SLRP) family. Members of this family are typically associated with the extracellular matrix. Opticin is present in significant quantities in the vitreous of the eye and also localizes to the cornea, iris, ciliary body, optic nerve, choroid, retina, and fetal liver. Opticin may noncovalently bind collagen fibrils and regulate fibril morphology, spacing, and organization. The opticin gene is mapped to a region of chromosome 1 that is associated with the inherited eye diseases age-related macular degeneration (AMD) and posterior column ataxia with retinosa pigmentosa (AXPC1).

Excerpted from Wikipedia’s “OPTC” article, available under the CC BY-SA 4.0 licence.

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