MIS18BP1
Sign in to saveAlso known as C14orf106, HSA242977, KNL2, M18BP1, MIS18 binding protein 1
MIS18 binding protein 1 is a protein that in humans is encoded by the MIS18BP1 gene. The gene is also known as LKNL2, M18BP1, C14orf106, and HSA242977. M18BP1 protein is an important component in the CENPA deposition pathway, and in most eukaryotes M18BP1 is necessary for chromosome segregation.
Gene data
MIS18BP1- Name
- MIS18 binding protein 1
- Type
- protein-coding
- Position
- 45,203,125–45,253,542 (−)
- Aliases
- C14orf106, HSA242977, KNL2, M18BP1
- Ensembl
- ENSG00000129534
- RefSeq RNA
- NM_018353.5, XM_005267833.6, XM_011536923.1, XM_017021425.2, XM_047431545.1
- RefSeq protein
- NP_060823.3, XP_005267890.1, XP_011535225.1, XP_016876914.1, XP_047287501.1
Predicted to enable DNA binding activity. Predicted to be involved in cell division. Predicted to be located in nucleoplasm. [provided by Alliance of Genome Resources, Apr 2022]
Gene Ontology
Biological process
Molecular function
Pathways
via MyGene.info
Gene · Ensembl
MIS18 binding protein 1
- Symbol
- MIS18BP1
- Biotype
- Protein coding
- Organism
- Homo sapiens
- Location
- 14:45,203,125-45,253,542
- Strand
- Reverse (−)
- Assembly
- GRCh38
via Ensembl · EMBL-EBI
Wikidata facts
- Instance of
- gene
- Image
- Protein C14orf106 PDB 1wgx.png
Show 8 more facts
- HomoloGene ID
- 10147
- found in taxon
- Homo sapiens
- exact match
- identifiers.org/ncbigene/55320
- genomic start
- 45672393
- chromosome
- human chromosome 14
- genomic end
- 45722743
- cytogenetic location
- 14q21.2
- expressed in
- rectum
Sources (3)
via Wikidata · CC0
~1 min read
Encyclopedic overview
2 sectionsContents
- References
- Further reading
MIS18 binding protein 1 is a protein that in humans is encoded by the MIS18BP1 gene. The gene is also known as LKNL2, M18BP1, C14orf106, and HSA242977. M18BP1 protein is an important component in the CENPA deposition pathway, and in most eukaryotes M18BP1 is necessary for chromosome segregation.
==References==
Excerpted from Wikipedia’s “MIS18BP1” article, available under the CC BY-SA 4.0 licence.