NLGN1
Sign in to saveAlso known as NL1, neuroligin 1
Neuroligin-1 is a protein that in humans is encoded by the NLGN1 gene.
In the Vinony graph
Vinony's link graph records 6 inbound references to NLGN1, and connects out to Q180686, human chromosome 3 and Ensembl genome database project.
It is catalogued under the topic Genes on human chromosome 3.
Vinony links it to 7 Wikipedia language editions.
Gene data
NLGN1- Name
- neuroligin 1
- Type
- protein-coding
- Position
- 173,396,284–174,294,372 (+)
- Aliases
- NL1, NLG1
- Ensembl
- ENSG00000169760
- RefSeq RNA
- NM_001365923.2, NM_001365924.2, NM_001365925.2, NM_001365926.2, NM_001365927.2
- RefSeq protein
- NP_001352852.1, NP_001352853.1, NP_001352854.1, NP_001352855.1, NP_001352856.1
This gene encodes a member of a family of neuronal cell surface proteins. Members of this family may act as splice site-specific ligands for beta-neurexins and may be involved in the formation and remodeling of central nervous system synapses. [provided by RefSeq, Jul 2008].
Gene Ontology
Biological process
Molecular function
Pathways
via MyGene.info
Gene · Ensembl
neuroligin 1
- Symbol
- NLGN1
- Biotype
- Protein coding
- Organism
- Homo sapiens
- Location
- 3:173,396,284-174,294,372
- Strand
- Forward (+)
- Assembly
- GRCh38
via Ensembl · EMBL-EBI
Wikidata facts
Show 5 more facts
- HomoloGene ID
- 56690
- exact match
- identifiers.org/ncbigene/22871
- genomic end
- 174294372
- genomic start
- 173114074
- cytogenetic location
- 3q26.31
via Wikidata · CC0
~1 min read
Encyclopedic overview
4 sectionsContents
- Interactions
- See also
- References
- Further reading
Neuroligin-1 is a protein that in humans is encoded by the NLGN1 gene.
This gene encodes a member of the neuroligin family of neuronal cell surface proteins. Neuroligin-1 acts as splice site-specific ligand for β-neurexins and has been shown to localize to the postsynaptic compartment at excitatory synapses and is involved in the formation and remodeling of central nervous system synapses.
Excerpted from Wikipedia’s “NLGN1” article, available under the CC BY-SA 4.0 licence.