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GeneQ18026512· pop 6· linked from 105 articles

Also known as GluN1, MRD8, NMDA1, NMDAR1, NR1, NMD-R1, glutamate ionotropic receptor NMDA type subunit 1, NDHMSR

Glutamate Receptor Ionotropic, [NMDA] subunit zeta-1 is a protein that in humans is encoded by the GRIN1 gene.

Gene data

GRIN1
Name
glutamate ionotropic receptor NMDA type subunit 1
Type
protein-coding
Position
137,138,346–137,168,756 (+)
Aliases
DEE101, GluN1, MRD8, NDHMSD, NDHMSR, NMD-R1, NMDA1, NMDAR1, NR1, hNR1
RefSeq RNA
NM_000832.7, NM_001185090.2, NM_001185091.2, NM_001437330.1, NM_001437331.1
RefSeq protein
NP_000823.4, NP_001172019.1, NP_001172020.1, NP_001424259.1, NP_001424260.1

The protein encoded by this gene is a critical subunit of N-methyl-D-aspartate receptors, members of the glutamate receptor channel superfamily which are heteromeric protein complexes with multiple subunits arranged to form a ligand-gated ion channel. These subunits play a key role in the plasticity of synapses, which is believed to underlie memory and learning. Cell-specific factors are thought to control expression of different isoforms, possibly contributing to the functional diversity of the subunits. Alternatively spliced transcript variants have been described. [provided by RefSeq, Jul 2008].

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Wikidata facts

Image
PDB 2nr1 EBI.jpg
Show 5 more facts
HomoloGene ID
7187
genomic end
137168756
genomic start
137139154
cytogenetic location
9q34.3
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Glutamate Receptor Ionotropic, [NMDA] subunit zeta-1 is a protein that in humans is encoded by the GRIN1 gene.

The protein encoded by this gene is a critical subunit of N-methyl-D-aspartate receptors, members of the glutamate receptor channel superfamily which are heteromeric protein complexes with multiple subunits arranged to form a ligand-gated ion channel. These subunits play a key role in the plasticity of synapses, which is believed to underlie memory and learning. The gene consists of 21 exons and is alternatively spliced, producing transcript variants differing in the C-terminus. The sequence of exon 5 is identical in vertebrates, with exon 5 splicing demonstrated in human, mouse and rat. Cell-specific factors are thought to control expression of different isoforms, possibly contributing to the functional diversity of the subunits.

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