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ProteinQ2620886· pop 17· linked from 103 articles

Huntingtin

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Also known as HTT, HD protein, huntington disease protein, Htt1

Huntingtin (Htt) is a human protein encoded by the HTT gene, also known as IT15 ("interesting transcript 15"). Pathogenic expansions in HTT (disease-causing repeat length increases) cause Huntington's disease (HD), and the protein has also been implicated in mechanisms of long-term memory storage.

Protein · UniProt

Huntingtin

Gene
HTT
Organism
Homo sapiens (Human)
Length
3,142 aa
Molecular mass
347,603 Da
Evidence
1: Evidence at protein level

May play a role in microtubule-mediated transport or vesicle function

3D-structureAcetylationApoptosisCytoplasmCytoplasmic vesicleDisease variantEndosomeIntellectual disability
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Swiss-Prot (reviewed) · via UniProt

Clinical Trials

15 registered

via ClinicalTrials.gov

~12 min read

Encyclopedic overview

11 sections
Contents
  • Gene
  • Structure
  • Function
  • Interactions
  • Clinical significance
  • Huntington's disease
  • Mitochondrial dysfunction
  • See also
  • References
  • Further reading
  • External links

Huntingtin (Htt) is a human protein encoded by the HTT gene, also known as IT15 ("interesting transcript 15"). Pathogenic expansions in HTT (disease-causing repeat length increases) cause Huntington's disease (HD), and the protein has also been implicated in mechanisms of long-term memory storage.

HTT is expressed in many tissues, with the highest levels in the brain. Expression is developmentally regulated and required for embryogenesis. Huntingtin normally consists of 3,144 amino acids and has a predicted mass of ~350 kDa, depending on the length of its polyglutamine tract. Polymorphisms in HTT alter the number of glutamine residues: the wild-type allele encodes 6–35 repeats, whereas pathogenic expansions in HD exceed 36, with severe juvenile cases reaching ~250 repeats. The name huntingtin reflects this association with disease; IT15 was its earlier designation.

Excerpted from Wikipedia’s “Huntingtin” article, available under the CC BY-SA 4.0 licence.